We´re Burning It Up!
Wed, August 27 2014 » Business » Comments Off
The federal government issued sixty contracts from 2009 to 2014 in efforts to build Healthcare.gov, the federal insurance marketplace. According to a report issued today by the inspector general (OIG) of the Department of Health and Human Services (HHS), the government had already paid out just under half a billion dollars by February 2014, five months after the beginning of open enrollment. The government is already under obligation for another $300 million, and the estimated value of the sixty contracts totals $1.7 billion. The OIG provided a summary of its findings:
The 60 contracts related to the development and operation of the Federal Marketplace started between January 2009 and January 2014. The purpose of the 60 contracts ranged from health benefit data collection and consumer research to cloud computing and Web site development. The original estimated values of these contracts totaled $1.7 billion; the contract values ranged from $69,195 to over $200 million. Across the 60 contracts, nearly $800 million has been obligated for the development of the Federal Marketplace as of February 2014. As of that date, CMS had paid nearly $500 million for the development of the Federal Marketplace to the contractors awarded these contracts.
A few familiar names appear on the list of contracts, such as Northrop Grumman and Lockheed Martin. Also appearing are CGI Federal, widely blamed for the botched roll out of the site last October, and Accenture Federal Services, which has taken over for CGI in hopes that this year’s open enrollment will go better than 2013.
More here: http://www.weeklystandard.com/blogs/feds-healthcaregov-could-cost-17-billion_803866.html
Tue, August 26 2014 » Business » Comments Off
Without former LAPD Det. Mark Fuhrman, whose book “Murder in Greenwich” is credited with fingering Skakel as Martha Moxley’s killer, the 1975 mystery would never have been reopened by Connecticut officials.
So why should that bother the Clintons?
Simple. Next on Furhman’s agenda is none other than the strange death of the Clintons’ Whitewater lawyer, Vincent Foster. Foster was found shot to death in a Virginia park July 20, 1993 and the death was officially ruled a suicide.
But five federal investigations into the episode raised more questions than they answered and left many observers suspecting a White House cover-up.
“I’ve been researching and working on that in my spare time for almost three years now,” Fuhrman told Fox News Channel last year.
“If he committed suicide, he didn’t do at Fort Marcy Park,” the Skakel sleuth told then-Fox host Paula Zahn. “If they want it to be a suicide, the people that brought him there and staged the scene are going to have to come forward and state where he was and how he committed suicide.
“That’s never going to happen,” Fuhrman insisted. “So it will be a murder – it will be a suspicious murder for all time now.”
Fuhrman also complained about the Foster crime scene to the New York Post.
“There was no brain matter, no skull fragments, not anything behind his head or blood on the vegetation around it,” the former LAPD cop said. “It was a sunny day, the light was good, yet there was nothing noted, nothing photographed.”
If Fuhrman follows up “Murder in Greenwich” with “Murder in Fort Marcy Park,” Mrs. Clinton may have to put her presidential plans on hold.
(You can see the source here)
So which other conspiracy has Mark Fuhrman tackled in the past?
The Kennedy assassination.
But unlike the Vince Foster case, Fuhrman believes that the official version of the incident could be correct.
Here is a video of him explaining why he thinks Lee Harvey Oswald could have acted as a lone gunman killing the 35th President of the United States:
Publisher Alfred Regnery calls it “ironic” and agent Lucianne Goldberg has called it “high school stuff.” Whatever the spin, it’s clear that Regnery, who published Mark Fuhrman’s bestselling Murder in Brentwood, has passed on the controversial O.J. trial detective’s proposal to do a book on the death of Clinton colleague Vince Foster and instead signed up a book on the case by author Dan Moldea. Goldberg has pulled Fuhrman’s proposal, which has been circulating to New York publishers, and told PW that they are developing a new proposal, possibly on another high-profile crime.
(Source: Fuhrman Wthdraws Vince Foster Proposal )
Regnery told PW he had been thinking about doing a book on the Foster case for some time, but felt that Fuhrman lacked the political-insider access needed to write it, so he rejected the proposal. Regnery didn’t negotiate the deal with Moldea’s agent, Ron Goldfarb, however, until after Fuhrman had submitted his proposal. . . . [Regnery] said he would still like to publish another book by Fuhrman, a suggestion Goldberg scoffed at.
(Source: From Fuhrman to Foster )
This reintroduction of Ruddy’s ideas followed the publication of another book, penned by veteran crime reporter Dan Moldea, titled A Washington Tragedy: How the Death of Vincent Foster Ignited a Political Firestorm. Moldea was approached in 1997 by Regnery Publishing House, a famously conservative group whose leadership was nevertheless impressed by Moldea’s published works, including an exhaustive critique of LAPD Detective Mark Fuhrman, another Regnery author. In researching the debacle created by and surrounding Foster’s death, Moldea found that the most oft-used conspiracy scenario could be traced back to Park Police Major Robert Hines, who shared the idea with Reed Irvine of (Accuracy in Media), and Christopher Ruddy, who was then at the New York Post. Moldea concludes, and Maj. Hines publicly maintains, that Hines incorrectly told Irvine and Ruddy “…that there is no exit wound in Foster’s head.” Moldea profers further: “I don’t think there was anything nefarious here; he was being approached by reporters and he wanted something to say.” Still, the “missing” exit wound claim continued to surface, propagated mainly by Christopher Ruddy and Joseph Farah.
(Source: Wikipedia )
In addition: Judge Andrew Napolitano agrees that the death of Vince Foster could easily be brought back up and become an issue was Hillary Clinton to run for the Presidency of the United States:
As a Fox News analyst, Mark Fuhrman has also commented on the George Zimmerman case:
So what do you think?
Here is Sean Hannity speaking about a few other scenarios surrounding the strange death of Vince Foster:
And in addition, you can listen to a one hour documentary on the case from The Corbett Report:
Tue, August 26 2014 » Conspiracies » Comments Off
Czech Republic lawmakers have decided upon a new strategy to keep their streets clean:
Threatening to publish public urination on Youtube.
Will it keep drunks on Friday and Saturday night from relieving themselves right there on a public road?
Will it eventually sink in once “best of videos” are being uploaded for everyone to see what their co-workers and family members are up to on the weekend?
In other words: Looking forward to the “live stream”.
Sat, August 23 2014 » Picture of the Day » Comments Off
By Samantha Craft of Everyday Asperger’s, March 2012
Albert Einstein is believed by some to have had Asperger’s syndrome
This is a non-official checklist created by an adult female with Asperger’s Syndrome who has a son with Asperger’s Syndrome. Samantha Craft holds a Masters Degree in Education. Samantha Craft does not hold a doctorate in Psychiatry or Psychology. She has a life-credential as a result of being a female with Asperger’s Syndrome and being a parent of a child with Asperger’s Syndrome. She has created this list in an effort to assist mental health professionals in recognizing Asperger’s Syndrome in females.
Suggested Use: Check off all areas that strongly apply to the person. If each area has 75%-80% of the statements checked, or more, then you may want to consider that the female may have Asperger’s Syndrome.
Section A: Deep Thinkers
1. A deep thinker
2. A prolific writer drawn to poetry
3. Highly intelligent
4. Sees things at multiple levels including thinking processes.
5. Analyzes existence, the meaning of life, and everything continually.
6. Serious and matter-of-fact in nature.
7. Doesn’t take things for granted.
8. Doesn’t simplify.
9. Everything is complex.
10. Often gets lost in own thoughts and “checks out.” (blank stare)
Section B: Innocent
3. Experiences trouble with lying.
4. Finds it difficult to understand manipulation and disloyalty.
5. Finds it difficult to understand vindictive behavior and retaliation.
6. Easily fooled and conned.
7. Feelings of confusion and being overwhelmed
8. Feelings of being misplaced and/or from another planet
9. Feelings of isolation
10. Abused or taken advantage of as a child but didn’t think to tell anyone.
Section C: Escape and Friendship
1. Survives overwhelming emotions and senses by escaping in thought or action.
2. Escapes regularly through fixations, obsessions, and over-interest in subjects.
3. Escapes routinely through imagination, fantasy, and daydreaming.
4. Escapes through mental processing.
5. Escapes through the rhythm of words.
6. Philosophizes continually.
7. Had imaginary friends in youth.
8. Imitates people on television or in movies.
9. Treated friends as “pawns” in youth, e.g., friends were “students,” “consumers,” “soldiers.”
10. Makes friends with older or younger females.
11. Imitates friends or peers in style, dress, and manner.
12. Obsessively collects and organizes objects.
13. Mastered imitation.
14. Escapes by playing the same music over and over.
15. Escapes through a relationship (imagined or real).
16. Numbers bring ease.
17. Escapes through counting, categorizing, organizing, rearranging.
18. Escapes into other rooms at parties.
19. Cannot relax or rest without many thoughts.
20. Everything has a purpose.
Section D: Comorbid Attributes
1. OCD (Obsessive Compulsive Disorder)
2. Sensory Issues (sight, sound, texture, smells, taste)
3. Generalized Anxiety
4. Sense of pending danger or doom
5. Feelings of polar extremes (depressed/over-joyed; inconsiderate/over-sensitive)
6. Poor muscle tone, double-jointed, and/or lack in coordination
7. Eating disorders, food obsessions, and/or worry about what is eaten.
8. Irritable bowel and/or intestinal issues
9. Chronic fatigue and/or immune challenges
10. Misdiagnosed or diagnosed with other mental illness and/or labeled hypochondriac.
11. Questions place in the world.
12. Often drops small objects
13. Wonders who she is and what is expected of her.
14. Searches for right and wrong.
15. Since puberty, has had bouts of depression.
16. Flicks/rubs fingernails, flaps hands, rubs hands together, tucks hands under or between legs, keeps closed fists, and/or clears throat often.
Section E: Social Interaction
1. Friends have ended friendship suddenly and without person understanding why.
2. Tendency to over-share.
3. Spills intimate details to strangers.
4. Raised hand too much in class or didn’t participate in class.
5. Little impulse control with speaking when younger.
6. Monopolizes conversation at times.
7. Bring subject back to self.
8. Comes across at times as narcissistic and controlling. (Is not narcissistic.)
9. Shares in order to reach out.
10. Sounds eager and over-zealous at times.
11. Holds a lot of thoughts, ideas, and feelings inside.
12. Feels as if she is attempting to communicate “correctly.”
13. Obsesses about the potentiality of a relationship with someone, particularly a love interest.
14. Confused by the rules of accurate eye contact, tone of voice, proximity of body, stance, and posture in conversation.
15. Conversation can be exhausting.
16. Questions the actions and behaviors of self and others, continually.
17. Feels as if missing a conversation “gene” or thought-“filter”
18. Trained self in social interactions through readings and studying of other people.
19. Visualizes and practices how she will act around others.
20. Practices in mind what she will say to another before entering the room.
21. Difficulty filtering out background noise when talking to others.
22. Has a continuous dialogue in mind that tells her what to say and how to act when in a social situations.
23. Sense of humor sometimes seems quirky, odd, or different from others.
24. As a child, it was hard to know when it was her turn to talk.
25. She finds norms of conversation confusing.
Section F: Finds Refuge when Alone
1. Feels extreme relief when she doesn’t have to go anywhere, talk to anyone, answer calls, or leave the house.
2. One visitor at the home may be perceived as a threat.
3. Knowing logically a house visitor is not a threat, doesn’t relieve the anxiety.
4. Feelings of dread about upcoming events and appointments on the calendar.
5. Knowing she has to leave the house causes anxiety from the moment she wakes up.
6. All the steps involved in leaving the house are overwhelming and exhausting to think about.
7. She prepares herself mentally for outings, excursions, meetings, and appointments.
8. Question next steps and movements continually.
9. Telling self the “right” words and/or positive self-talk doesn’t often alleviate anxiety.
10. Knowing she is staying home all day brings great peace of mind.
11. Requires a large amount of down time or alone time.
12. Feels guilty after spending a lot of time on a special interest.
13. Uncomfortable in public locker rooms, bathrooms, and/or dressing rooms.
14. Dislikes being in a crowded mall, crowded gym, or crowded theater.
Section G: Sensitive
1. Sensitive to sounds, textures, temperature, and/or smells when trying to sleep.
2. Adjusts bedclothes, bedding, and/or environment in an attempt to find comfort.
3. Dreams are anxiety-ridden, vivid, complex, and/or precognitive in nature.
4. Highly intuitive to others’ feelings.
5. Takes criticism to heart.
6. Longs to be seen, heard, and understood.
7. Questions if she is a “normal” person.
8. Highly susceptible to outsiders’ viewpoints and opinions.
9. At times adapts her view of life or actions based on others’ opinions or words.
10. Recognizes own limitations in many areas daily.
11. Becomes hurt when others question or doubt her work.
12. Views many things as an extension of self.
13. Fears others opinions, criticism, and judgment.
14. Dislikes words and events that hurt animals and people.
15. Collects or rescues animals. (often in childhood)
16. Huge compassion for suffering.
17. Sensitive to substances. (environmental toxins, foods, alcohol, etc.)
18. Tries to help, offers unsolicited advice, or formalizes plans of action.
19. Questions life purpose and how to be a “better” person.
20. Seeks to understand abilities, skills, and/or gifts.
Section H: Sense of Self
1. Feels trapped between wanting to be herself and wanting to fit in.
2. Imitates others without realizing.
3. Suppresses true wishes.
4. Exhibits codependent behaviors.
5. Adapts self in order to avoid ridicule.
6. Rejects social norms and/or questions social norms.
7. Feelings of extreme isolation.
8. Feeling good about self takes a lot of effort and work.
9. Switches preferences based on environment and other people.
10. Switches behavior based on environment and other people.
11. Didn’t care about her hygiene, clothes, and appearance before teenage years and/or before someone else pointed these out to her.
12. “Freaks out” but doesn’t know why until later.
13. Young sounding voice
14. Trouble recognizing what she looks like and/or has occurrences of slight prosopagnosia (difficulty recognizing or remembering faces).
Section I: Confusion
1. Had a hard time learning others are not always honest.
2. Feelings seem confusing, illogical, and unpredictable. (self’s and others’)
3. Confuses appointment times, numbers, or dates.
4. Expects that by acting a certain way certain results can be achieved, but realizes in dealing with emotions, those results don’t always manifest.
5. Spoke frankly and literally in youth.
6. Jokes go over the head.
7. Confused when others ostracize, shun, belittle, trick, and betray.
8. Trouble identifying feelings unless they are extreme.
9. Trouble with emotions of hate and dislike.
10. Feels sorry for someone who has persecuted or hurt her.
11. Personal feelings of anger, outrage, deep love, fear, giddiness, and anticipation seem to be easier to identify than emotions of joy, satisfaction, calmness, and serenity.
12. Situations and conversations sometimes perceived as black or white.
13. The middle spectrum of outcomes, events, and emotions is sometimes overlooked or misunderstood. (All or nothing mentality)
14. A small fight might signal the end of a relationship or collapse of world.
15. A small compliment might boost her into a state of bliss.
Section J: Words and Patterns
1. Likes to know word origins.
2. Confused when there is more than one meaning to a word.
3. High interest in songs and song lyrics.
4. Notices patterns frequently.
5. Remembers things in visual pictures.
6. Remembers exact details about someone’s life.
7. Has a remarkable memory for certain details.
8. Writes or creates to relieve anxiety.
9. Has certain “feelings” or emotions towards words.
10. Words bring a sense of comfort and peace, akin to a friendship.
(Optional) Executive Functioning This area isn’t always as evident as other areas
1. Simple tasks can cause extreme hardship.
2. Learning to drive a car or rounding the corner in a hallway can be troublesome.
3. New places offer their own set of challenges.
4. Anything that requires a reasonable amount of steps, dexterity, or know-how can rouse a sense of panic.
5. The thought of repairing, fixing, or locating something can cause anxiety.
6. Mundane tasks are avoided.
7. Cleaning may seem insurmountable at times.
8. Many questions come to mind when setting about to do a task.
9. Might leave the house with mismatched socks, shirt buttoned incorrectly, and/or have dyslexia.
10. A trip to the grocery store can be overwhelming.
11. Trouble copying dance steps, aerobic moves, or direction in a sports gym class.
12. Has a hard time finding certain objects in the house, but remembers with exact clarity where other objects are.
This list was compiled after nine years of readings, research, and experience associated with Asperger’s Syndrome. More information can be found at http://aspergersgirls.wordpress.com © Everyday Aspergers, 2012 This non-official checklist can be printed for therapists, counselors, psychiatrists, psychologists, professors, teachers, and relatives, if Samantha Craft’s name and contact information remain on the print out.
Wed, August 20 2014 » Science » Comments Off
A large piece of interest regarding ancestry of today´s rh negative populations worldwide revolves around whether or not certain genetic markers are more likely to to be those of rh negative individuals than others.
My answer despite loads of criticism:
Of course there is a connection between ancestry and blood type. Wouldn´t you agree that there is less of a chance that someone with the East Asian haplogroup O3 (East Asian countries all have less than 1 percent rh negatives)
is less likely to be RH- than someone sporting the Celtic marker R1b (M-269)?
Any suggestion otherwise would be highly illogical.
And at the same token, as different locations of Europe have different percentages of rh negative populations,
so do those regions vary in terms of how many people have certain genetic markers.
Now the question is how we can trace everything back and if there was one original tribe, haplogroup subclade to be specific, where everyone was a member of one specific marker and everyone within that tribe was 100% rh negative.
Even though I disagree with several analogies made by Italian population geneticist Luigi Luca Cavalli-Sforza, I cannot help but pay attention to his claim that the original Basques were 100 percent rh negative and that the current presence of rh positive blood as well as blood type B and therefore AB
were “imported” through mixing with their neighbors.
This very statement screams for an examination as to who the Basques are, who the original Basques were and
which frequencies of genetic markers now and then can trace us back to their origin which may or may not lie in a different region than the Pyrenees Mountain Range which they are currently populating.
Having communicated with several Basque individuals via Facebook and Twitter, I have learned that to many Basques their own origin is in fact a mystery and the fact Basques could easily be the “first Europeans” dating back as far as 7,000 to 13,000 years back
would explain how passing on history could have gotten lost in translations.
So there are two things that we can look at when searching for the roots of the Basques:
1) Language and languages with similarities.
2) Genetic marker frequencies amongst the Basques in comparison with other parts of the world where the percentage is high.
Dené–Caucasian is a proposed broad language family that includes the Sino-Tibetan, North Caucasian, Na-Dené, Yeniseian, Vasconic (including Basque) and Burushaski language families. A connection specifically between Na-Dené and Yeniseian was proposed by Edward Vajda in 2000, and has met with some acceptance.
As the above map highlight, the Pyrenees Mountain region is the only spot in all of Europe where the language family is being highlighted and it is also present within the Fertile Crescent.
So let´s look at genetic markers:
Initially I have found a study where 5,000 year old Basque graves were being examined and around 15 percent
had haplogroup J as in J1c1 and J2a (also known as the Cohen marker), meaning the further you go back
within the Basque population, the higher the percentage of haplotype J becomes:
This study provides a more complete characterization of the mitochondrial genome variability of the Basques, including data on the hypervariable segment HVII of the D-loop region, which remains relatively unknown. To that end, genomic DNA from 55 healthy men living in the Arratia Valley (Biscay province) and the Goiherri region (Guipúzcoa province) was examined by direct sequencing. Three-generation pedigree charts were compiled to ensure the collection from autochthonous individuals. The most notable findings emerging from the analysis of haplogroup composition are: (i) lack of U8a mitochondrial lineage, a rare subhaplogroup recently identified in Basques and proposed as a Paleolithic marker, (ii) low frequency of haplogroup V, which conflicts with results of earlier analyses describing high frequencies in southwestern Europe, and (iii) high frequency of haplogroup J, especially subhaplogroups J1c1 and J2a. The frequency of haplogroup J does not coincide with previous mtDNA studies in present-day Basques, but is congruent with frequencies found in prehistoric and historic Basque populations. In explaining divergence in haplogroup composition between modern Basque samples, we hypothesized spatial heterogeneity promoted by population fragmentation due to extreme limitation of dispersal opportunities during the Pleistocene glaciations. Similarities between extinct and extant Basque populations as for the high frequency of lineage J, as well as the abundance of this haplogroup in northern Spain endorse a shift in the focus of attention of mtDNA analysts. A refined dissection of haplogroup J might provide more solid evidence about the process of postglacial recolonization of Europe, and thus about the shaping of the European gene pool.
But there is more:
mtDNA sequence variation was studied in 121 dental samples from four Basque prehistoric sites, by high-resolution RFLP analysis. The results of this study are corroborated by (1) parallel analysis of 92 bone samples, (2) the use of controls during extraction and amplification, and (3) typing by both positive and negative restriction of the linked sites that characterize each haplogroup. The absence of haplogroup V in the prehistoric samples analyzed conflicts with the hypothesis proposed by Torroni et al., in which haplogroup V is considered as an mtDNA marker for a major Paleolithic population expansion from southwestern Europe, occurring approximately 10,000-15,000 years before the present (YBP). Our samples from the Basque Country provide a valuable tool for checking the previous hypothesis, which is based on genetic data from present-day populations. In light of the available data, the most realistic scenario to explain the origin and distribution of haplogroup V suggests that the mutation defining that haplogroup (4577 NlaIII) appeared at a time when the effective population size was small enough to allow genetic drift to act-and that such drift is responsible for the heterogeneity observed in Basques, with regard to the frequency of haplogroup V (0%-20%). This is compatible with the attributed date for the origin of that mutation (10,000-15, 000 YBP), because during the postglacial period (the Mesolithic, approximately 11,000 YBP) there was a major demographic change in the Basque Country, which minimized the effect of genetic drift. This interpretation does not rely on migratory movements to explain the distribution of haplogroup V in present-day Indo-European populations.
Haplogroup V derives from the HV0a. In 1998 it was argued that V spread over Europe from an Ice Age refuge in Iberia. However more recent estimates of the date of V would place it in the Neolithic. That makes an origin in the Near East more plausible.
Haplogroup V is a relatively rare mtDNA haplogroup found in approximately 4% of native Europeans. Its highest concentration is among the Saami people of northern Scandinavia (approximately 59%), where its divergence time is estimated at 7600 YBP (years before present). It has been found at approximately 10% among the Maris of the Volga-Ural region, leading to the suggestion that this region might be the source of the V among the Saami.
Haplogroup V is also found at higher than average levels in Cantabrian people (15%) of northern Iberia, and somewhat lower in nearby Basque people (10.4%). It also is found in particularly high concentrations (16.3%) among the Berbers of Matmata, Tunisia.
Above images from Matmata, the small Berber speaking town in southern Tunisia
The connection between Basques and Berbers is crucial as both groups top the list of rh negative populations today.
So let´s take a look at the haplogroups strongly and in some cases uniquely represented within populations known to represent rh negative individuals at the highest percentages in the world:
Haplogroup J makes up almost 9% of mtDNAs in Europe and ∼13% in the Near East. The geographic distribution of its subclades supports the view that haplogroup J initially diversified in the Near East. A single lineage from the North Caucasus, defined only by the recurrent HVS-I transition at np 16193, branches immediately before J1d in our tree, and may represent a paraphyletic lineage within J1. If so, it would be the only lineage found not to fall within one of the five major subclades of haplogroup J; possible related lineages in the control-region database are found in Turkey, close to the likely source area for J1.
Our analysis confirms that haplogroups J and T and their major subclades (J1 and J2, T1 and T2) most likely arose in the Near East between the time of first settlement by modern humans and the LGM. The overall age estimate for JT is very similar to that for the second basal haplogroup R clade in western Eurasia, haplogroup U, at ∼58 ka ago. Both date to the beginning of the pluvial marine isotope stage (MIS) 3, at which point haplogroup R lineages emerging in the vicinity of the Gulf Oasis on the southern route out of Africa are likely to have begun their dispersal north into the Fertile Crescent. It seems plausible to regard JT and U as belonging to members of the same early human group, ancestral to both Near Easterners and Europeans.
From the genetic point of view, it has been suggested that the second principal component of classical marker variation in Europe may have been formed by the Late Glacial expansion,12 and there has been evidence from mtDNA for human expansions from the Franco-Cantabrian refugium; namely, signatures in haplogroups V,12,13 H1, H3,14,15 H5,16 and U5b1b17,18 as well as in haplogroup U5b3, indicating dispersal from the Italian Peninsula,19 and in U4 and U5a, indicating expansion from the East European Plain.20,21 As for the male-specific region of the Y chromosome (MSY), previous studies suggested that the European frequency clines associated with R1a-M17 and R1b-M173 (xM17) (now called R1b-M269) might have been due to population expansions from Ukrainian and Franco-Cantabrian refugia, respectively.22–24 Although a more complex scenario is emerging from the progressive improvement of the Y chromosome phylogeny,25–29 signs of post-LGM re-expansions from eastern European, Iberian, and southern Balkan refugia are suggested by the distribution of some R (R1a-M458, R1b-M412)25,27 and I (I-M423)30 subclades, respectively. By contrast, the majority of mtDNA haplogroups J and T131,32 and Y chromosome haplogroup J,22 along with G2a and E1b1b1a1b (on the basis of ancient-DNA studies),33–35 have been proposed as candidates for markers of the Neolithic expansion into Europe from its origin in the Fertile Crescent of the Near East.
Today, Jonas has shared an article called Do We Choose Our Friends Because They Share Our Genes? and the claim in a nutshell is:
People often talk about how their friends feel like family. Well, there’s some new research out that suggests there’s more to that than just a feeling. People appear to be more like their friends genetically than they are to strangers, the research found.
I have often asked:
Who do you feel closest to?
Could that feeling have something to do with common ancestry?
And would people with the same blood type be similar to one another based on ancestry and there being an original group which once has represented that specific type within the population both you and whoever you feel familiar with had ancestry within?
It is easy to dismiss any of the above.
But for some reason this topic keeps people coming back regaining interest as they get to know their friends´ blood types.
So ask yourself:
Assuming you have a brother or sister you have never met and meet that person accidentally, do you believe that you would sense similarity?
How about a cousin?
The same way family members share genetic markers with us, could it be that those sharing broader genetic markers with us could seem more familiar to us?
Blood types and genetic ancestries do connect and the question is to what degree.
And if in fact there is a strong common ancestry amongst us dating back to where haplogroups J and T originate, the ancient civilization of Mesopotamia could have been constructed by rh negative people responsible for the many discoveries and invention during that time in that region.
First modern medicine was practiced. Calculus was first applied. A banking system was in place and yes … beer was likely first discovered and introduced there.
The widely promoted theory that Neanderthals were rh negative now has been proven to be false and the first mutation into rh negative blood could have easily occurred in Africa with those carrying it migrating towards the Fertile Crescent from there.
So whatever stories you have been told (and most of them are false), it is important to distance ourselves from other people´s opinions and fabrications, yet hold on to what we ourselves truly sense and believe. Even if we are not able to place any fitting words around it and even if others try to tell us that we are simply imagining things.
And most of all, we need to protect ourselves and our own energy.
As advanced as Mesopotamia, Sumer and Babylon were, how did those civilizations end up?
Should we lower our personal level to keep from being destroyed?
The answer is no. To be more specific: Unity amongst all Rh Negative people!
Because regardless where our common ancestry lies, the fact that we share a common ancestry makes us family. An extended family. And as we are scattered throughout this world, we are now able to find one another again through the power of the web. Or the many groups forming such as this one:
How far have you traced back your ancestry?
Have you had a genetic test done?
What were the results?
Mon, August 18 2014 » Blood Type Studies » Comments Off
Here is a collection of videos of animals playing which I have seen over the past few months. Some of the videos are hosted on Facebook, so click the image to view them there:
Guitar Playing Dog
Dees Plays in Puddle
Dog Jumps From Tree
Goat Pushed Angler Into River
The Fearless Raven
Mon, August 18 2014 » Animals » Comments Off
The following map demonstrates very well why nationality matters none. All nations except a few have changed borders to such a high degree of frequency that the populations are a mix of tribes coming together based on borders developed by governments. What is your true origin and how far have you traced back your ancestry?
Sun, August 17 2014 » Business » Comments Off
Click on the image to watch the video on Facebook:
Sun, August 17 2014 » Animals » Comments Off
Ancestral DNA testing could do more than reveal the identities of distant cousins or whether you descended from Genghis Khan.
Doctors could one day be using your unique ancestral history to diagnose and treat you better. So says a recent study published in Science, in which researchers from Stanford University, UC San Francisco and several other institutions investigated variations in Mexican ancestry. Some indigenous groups in Mexico are as genetically different as Europeans are from Chinese, making mestizos — most Mexicans — who have indigenous, European and African blood, wildly diverse. And those differences between individuals could impact the way doctors interpret even the most seemingly straightforward data.
The new findings suggest that physicians could sequence their patients’ DNA to precisely determine their genetic background and adjust clinical guidelines accordingly, versus the more common practice of factoring in race or ethnicity, as doctors do for kidney function tests, prostate cancer screenings and more. And in the U.S., doctors use only three categories: Mexican, African American or Caucasian, Burchard said. Not only could DNA sequencing prevent underdiagnoses, it would also save money and time otherwise spent on treating patients who may actually be healthy for someone with their genetic ancestry.
Racial categories like Asians and Latinos are largely based on physical features and culture — factors shaped by political, social and other reasons — not genetic differences. Populations even within a geographic region — like Mexico — show enormous genetic diversity.
An increasingly multiracial world might only worsen the problems of traditional race-based clinical guidelines. “How would you classify Obama?” asks Esteban González Burchard, a professor in the UCSF schools of Pharmacy and Medicine and study co-author. The president has a Kenyan father and white mother — so which race’s clinical guidelines would doctors use? Typically, a receptionist guesses patients’ race when they arrive for an appointment and relays that information to a technician, who then notifies the physician, Burchard said.
“We’re moving beyond blanket definitions like Mexican or Latino,” said Andrés Moreno-Estrada, a life sciences research associate at Stanford and the study’s lead author. “Now we’re putting finer details on that map.”
Some worry that the medical field’s focus on genetics might eclipse research on other factors that influence health. “The effects of social class on lung function have been largely ignored in favor of the focus on race and ethnic difference,” said Lundy Braun, a professor of pathology and laboratory medicine, as well as Africana studies, at Brown University. Poverty increases the risk of a host of health problems. And poorer neighborhoods have less access to parks and grocery stores that sell fresh produce.
But it’s undeniable: genes play a major role in health, even if they do so alongside other factors. The problem is that, to date, most large-scale genetic analyses have focused on European populations. “Very few studies have been done on Hispanics or Latinos,” Burchard said. So he and a team of researchers examined the DNA of more than 500 people from remote indigenous villages and cosmopolitan centers throughout Mexico, as well as Mexican Americans in California.
Mexico’s genetic tapestry turned out to be richer than they had imagined. The team’s analysis revealed that the country’s indigenous populations diverge genetically along a northwest-to-southeast axis, with variations that grow starker with distance. That means a Seri from the Gulf of California and a Lacandon Maya near the southern border are as genetically different as Europeans from Chinese.
But most people of Mexican descent aren’t indigenous, but mestizo. Would their DNA still bear the genetic signatures of their Mayan, Aztec or other indigenous ancestors, or did centuries of mixing with Spanish conquistador genes erase them forever? Surprisingly, the mestizos studied tended to have the same genetic markers as indigenous populations nearby — closely replicating the geographic differences found in indigenous DNA samples.
The researchers then examined whether the results of a lung function test (used to diagnose asthma and other respiratory problems) from kids in Mexico City and the San Francisco Bay Area reflected Mexico’s genetic diversity. (Burchard is a pulmonologist at UCSF.) Sure enough, the lungs of a mestizo with western indigenous ancestry could appear a decade younger than an equally healthy person of the same age from Yucatán, who likely has Eastern indigenous blood. That means doctors should be using different criteria to diagnose lung disease for each population.
These findings could lay the groundwork for devising precise screenings or even treatments, and guide decisions about how to allocate resources for research and care. Researchers could apply the same techniques to other regions and diagnostic tools besides lung function tests. Burchard and his colleagues published a study in 2010 correlating the amount of African ancestry participants had to their lung function — but at a much smaller scale.
The new Science study also highlights evolutionary history’s crucial, yet oft-overlooked role in biomedical research. Most population geneticists investigate patterns of human migration and variation — projects that typically receive scant funding compared to those with direct medical applications. But clinically relevant findings “are often rooted in studies that were simply curious about evolution,” said Noah Rosenberg, an evolutionary biologist at Stanford.
In the end, both medicine and evolutionary biology have unearthed the same complex genetic diversity from thousands of years of human wandering, settling, mixing. “This is driving the ball down the field toward precision medicine,” Burchard said — beyond black-and-white diagnoses.
Source: USA Today